AKI assessment - How do patients present?

  • Not usually a problem unless the patient has a reduced conscious level (a late sign of serious organ failure).

  • The patient may present with:

    • Raised respiratory rate and/ or breathlessness. This can be due to compensation for metabolic acidosis or may be due to fluid overload.

    • On auscultation: bi-basal fine crackles or reduced breath sounds at the bases if developing fluid overload.

  • The patient may present:

    • hypotensive and tachycardic as a result of hypovolaemia/ shock

    • Other signs of this will include dry mucous membranes, cool peripheries, delayed CRT, decreased skin turgor

    • The patient may feel thirsty

    • Peripheral oedema if developing fluid overload

    • low urine output, concentrated urine

  • The patient may present:

    • drowsy or with new confusion due to the accumulation of urea or drugs

    • headaches or lightheadedness due to hypovolaemia

    • May have a distended/ palpable bladder if outflow obstruction

    • May present with abdominal or flank pain if presenting with intrinsic renal pathology